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Home / Encyclopedia / Recovery & Healing / Pentadeca Arginate
MONOGRAPH No. 101
TYPE
Pentadecapeptide (BPC-157 arginate salt)
MW
~1.5 kDa (peptide)
CAS
Not assigned
EU STATUS
Not approved · Research
WADA
Not listed
MIN PURITY
≥98% HPLC
🧬 Recovery & Healing

Pentadeca Arginate — EU research guide.

Pentadeca Arginate (PDA) is the arginate-salt form of BPC-157 — the same 15–amino-acid sequence bound to arginine instead of acetate. The modification is claimed to improve stability and shelf life while retaining BPC-157's tissue-repair profile.

Last reviewed:
Key findings at a glance
  • Same 15-amino-acid sequence as BPC-157, prepared as an arginate (not acetate) salt.
  • The arginate salt is claimed to improve stability and shelf life; the active peptide is unchanged.
  • No independent human clinical trials of PDA specifically — evidence is the BPC-157 animal literature.
  • Claims of superiority over standard BPC-157 rest on the stability argument, not comparative data.
  • Not approved by any regulator; sold as a research compound only.

What is Pentadeca Arginate?

Pentadeca Arginate (PDA) is not a new molecule in the strict sense — it is BPC-157 (a 15-amino-acid "Body Protection Compound" fragment originally identified in gastric juice) prepared as an arginate salt rather than the more common acetate salt. "Pentadeca" refers to the fifteen residues; "arginate" refers to the arginine counter-ion. The active peptide sequence is identical to BPC-157; what changes is the salt form, which vendors claim improves stability in acidic conditions and shelf life.

How is it different from BPC-157?

Functionally, PDA and BPC-157 share the same proposed mechanisms: promotion of angiogenesis (new blood-vessel formation), modulation of the nitric-oxide system, collagen synthesis support, and anti-inflammatory signalling without fully suppressing the repair response. The main practical difference is the counter-ion. Some vendors market the arginine component as providing additional nitric-oxide benefit, but at the molar quantities present in a typical microgram-scale peptide dose, the independent arginine contribution is pharmacologically minor. The realistic distinction is salt stability, not a different drug.

What does the research show?

There are no large human clinical trials of PDA specifically. The evidence base is the BPC-157 literature — predominantly rodent studies showing accelerated tendon-to-bone healing, ligament and muscle repair, and gastrointestinal protection — plus anecdotal community reports. PDA inherits this evidence by virtue of being the same peptide; it does not have an independent human trial record of its own as of mid-2026. Claims of superiority over standard BPC-157 rest on the stability argument, not on comparative clinical data.

EU legal status

Not approved as a medicine in the EU. PDA occupies the same unscheduled "research chemical" position as BPC-157 — not specifically prohibited by name in most member states, but not legal for human use, sale as a medicine, or marketing with therapeutic claims. It is sold by research-peptide vendors as a laboratory compound.

Research compound. Not EMA/FDA-approved. No human use outside authorised research settings. PDA is the same active sequence as BPC-157 in a different salt form — not a separately validated drug.
✓ PeptideCompare tracks Pentadeca Arginate and BPC-157 from EU research vendors with COA documentation, so you can compare purity and salt form before sourcing.

Molecular information

Molecular weight
~1.5 kDa (peptide)
CAS number
Not assigned

Pharmacokinetics

No established human pharmacokinetic data. Published human PK parameters for this compound are not available; reported data are limited to animal models or absent. No curve is shown, to avoid implying data that does not exist.

Pentadeca Arginate across EU suppliers

COA-verified EU vendors · Updated monthly

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