PeptideCompareAdamax — EU research guide.
Adamax is a combination research compound typically blending Semax and Selank in a single preparation, designed for concurrent nootropic, anxiolytic and BDNF-stimulating research in animal models.
What is Adamax?
Adamax is not a single molecular entity but a pre-formulated blend of Semax (ACTH 4–7 Pro-Gly-Pro) and Selank (Thr-Lys-Pro-Arg-Pro-Gly-Pro). It is sold by EU research vendors as a convenience preparation for researchers studying concurrent BDNF upregulation (Semax) and GABAergic anxiolysis (Selank). Individual component ratios vary by vendor.
What does the research show?
The evidence base for Adamax as a combined entity is absent from peer-reviewed literature — all mechanistic data derives from individual Semax and Selank research. The theoretical rationale is synergy between the nootropic (Semax) and anxiolytic (Selank) axes, reducing stress-related cognitive interference while simultaneously upregulating neurotrophic factors.
EU legal status
As a blend of two research compounds, Adamax carries the same regulatory status as its components — not approved anywhere, available for laboratory research only. Purity verification of both components independently is recommended.
Molecular information
Pharmacokinetics
Potency retained over time
Each line is one storage condition: it starts at 100% and falls as potency is lost. Above the dashed 80% line the material is considered reliable — hover for exact values.
⚠ Modelled per compound from molecular structure — chain length, cyclisation, oxidation- and deamidation-prone residues and light sensitivity. Indicative only, not lab-measured per batch.
Adamax across EU suppliers
COA-verified EU vendors · Updated monthly
References
- Rat study of Semax, not of Adamax: a single intranasal application raised hippocampal BDNF protein up to about 1.4-fold and the animals did better in a conditioned avoidance task. Animal data on one parent peptide only. Dolotov OV, Karpenko EA, et al. Semax, an analog of ACTH(4-10) with cognitive effects, regulates BDNF and trkB expression in the rat hippocampus. Brain Res. 2006;1117(1):54-60. DOIPubMed 16996037
- Rat gene-expression study of Selank, not of Adamax, using real-time PCR: 45 genes changed one hour after administration, with an overlap with the effect of GABA itself. Animal, mechanistic data only. Volkova A, Shadrina M, Kolomin T, Andreeva L, Limborska S, Myasoedov N, Slominsky P. Selank administration affects the expression of some genes involved in GABAergic neurotransmission. Front Pharmacol. 2016;7:31. DOI